Transforming Growth Factor-j3 in Cardiac Ontogeny and Adaptation

نویسندگان

  • W. Robb MacLellan
  • Thomas Brand
  • Michael D. Schneider
چکیده

on transcriptional activation by serum response factor. A truncated form of the type II TGF-j3 receptor, created by deletion of the cytoplasmic kinase domain, acts as a dominant suppressor of TGF-f3 signal transduction in cultured cardiac muscle cells and may provide a suitable means to establish the functions of TGF-f3 in vivo. (Circ Res. 1993;73:783-791.)

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Ontogeny and Adaptation

on transcriptional activation by serum response factor. A truncated form of the type II TGF-j3 receptor, created by deletion of the cytoplasmic kinase domain, acts as a dominant suppressor of TGF-f3 signal transduction in cultured cardiac muscle cells and may provide a suitable means to establish the functions of TGF-f3 in vivo. (Circ Res. 1993;73:783-791.)

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تاریخ انتشار 2005